24-002750

Submitted by fratecm on
Post Date/ Solicitation Issue Date
Closing Response Date
Proposed Award Date
Project Title
Copeptin (proAVP) measurement in plasma
Contracting Office
National Institute of Mental Health (NIMH)

Contact Points

Primary Contract Specialist

Cheryl
Leone
cleone@mail.nih.gov

Secondary Contracting Officer

Dionne
Draper
dionne.draper@nih.gov
NAICS Code Number
621511
Medical Laboratories
FPDS Classification Code
B504
Delivery of Goods
30 days ARO
Competition Status
Non-Competitive
Vendor Name
Mayo Clinic - Mayo Medical Laboratories
Vendor Address
3050 Superior Dr NW
Rochester, MN 55901
Single-Sole Source Determination
Intensive search was made for vendors who can process the measurement of copeptin. The Mayo Clinic Lab has a proven reputation for leading innovation with excellence in lab tests for healthcare and research needs. They are specialized at measuring copeptin proAVP with homogeneous automated immunofluorescent assay on the BRAHMS Kryptor Compact PLUS system.
Background/Description of Requirement

The purpose is to measure copeptin levels in 210 clinical plasma samples to study its association with suicidal thoughts and antisuicidal response to ketamine.

Our lab has been doing basic and clinical studies with mood disorders to investigate the biomarkers and mechanisms underlying ketamine’s rapid antidepressant effects. Vasopressin (AVP) is a neuropeptide molecule classically known as an antidiuretic hormone, secreted by the posterior pituitary in response to changes in plasma osmolality or blood pressure. It also plays a role in regulating HPA axis activity under acute and chronic stress conditions. Copeptin is a byproduct of the cleaving of AVP precursor peptide. Our collaborators at the Mayo Clinic are specialized at measuring copeptin levels using the BRAHMS-Kryptor Copeptin Assay.

Project Background.  The NIMH Experimental Therapeutics & Pathophysiology Branch has been doing basic and clinical studies related to depression and treatment outcomes with rapid antidepressant agents. Under chronic stress, adrenocorticotropic hormone (ACTH) release is stimulated primarily by AVP to maintain physiological homeostasis even after corticotrophin-releasing hormone (CRH) release is suppressed via negative feedback from hypercortisolemia. Multiple studies found social stress-induced increases in AVP, suggesting a role for AVP-driven ACTH release and HPA axis activation in social stress. Genome-wide association studies also revealed that genetic polymorphisms in V1b receptors were associated with childhood-onset mood disorders. Findings to date have been mixed, partly owing to methodological barriers associated with AVP’s pulsatile secretion and rapid plasma secretion. Copeptin is a byproduct of the cleaving of AVP precursor peptide. Owing to its stability at room temperature, plasma copeptin has been increasingly researched as a surrogate biomarker for plasma AVP. To better understand the role of AVP in mood disorders and stress-related pathophysiology, studies with reliable copeptin measurement are needed.

Interested parties may identify in writing their interest and capability in response to this requirement. Responses to this notice shall contain sufficient information to establish the interested parties’ bona-fide capabilities for fulfilling the requirement and include: unit price, list price, shipping and handling costs, the delivery period after contract award, the prompt payment discount terms, the F.O.B. Point (Destination or Origin), the Dun & Bradstreet Number (DUNS), the Taxpayer Identification Number (TIN), and the certification of business size. All offerors must have an active registration in the System for Award Management (SAM) www.sam.gov.

All responses must be received by closing date and must reference the announcement. Responses may be submitted electronically to the attention of the contract specialist. Fax responses will not be accepted.

All responsible sources may submit a bid, proposal, or quotation which shall be considered by the agency.